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FDA ApprovedFDA Approved

SS-31 (Elamipretide)

Mitochondrial-Targeted Peptide | Cardiolipin Protector

SS-31, also known as Elamipretide or Bendavia, is a small aromatic-cationic tetrapeptide that selectively targets mitochondria. Unlike traditional antioxidants, SS-31 specifically binds to cardiolipin in the inner mitochondrial membrane, protecting it from oxidative damage and improving cellular energy production. This unique mechanism makes it particularly promising for age-related conditions, heart disease, and mitochondrial disorders.

Daily dose

5-40mg

Frequency

Once daily

Cycle length

4-12 weeks

Storage

2-8°C (refrigerated)

Key benefits

Directly protects mitochondria, improves cellular energy, reduces oxidative damage, enhances exercise capacity

How it works

Selectively binds to cardiolipin in inner mitochondrial membrane, preventing lipid peroxidation and optimizing electron transport chain function

Dosage protocols

Goal

General Mitochondrial Support

Dose

5-10mg · Once daily

Route

Subcutaneous

Goal

Athletic Performance

Dose

10-20mg · Once daily pre-workout

Route

Subcutaneous

Goal

Clinical Protocols

Dose

40mg · Once daily

Route

Subcutaneous or IV

Goal

Acute Cardioprotection

Dose

0.25mg/kg/hr · Continuous infusion

Route

Intravenous

Research indications

mitochondrial

Enhanced ATP ProductionImproves mitochondrial efficiency and cellular energy generation
Oxidative Stress ReductionProtects against reactive oxygen species at the source
Mitochondrial Disease SupportShows promise for various mitochondrial disorders

cardiovascular

Cardiac ProtectionReduces ischemia-reperfusion injury and improves heart function
Heart Failure SupportMay improve cardiac energetics in heart failure patients
Exercise ToleranceEnhances cardiac output and exercise capacity

anti Aging

Muscle FunctionReverses age-related decline in muscle mitochondria
Cognitive ProtectionMay protect against age-related cognitive decline
Cellular RejuvenationRestores youthful mitochondrial function

Administration

injectable
topical

Interactions

Synergistic
NAD+Both support mitochondrial function through different mechanisms - SS-31 protects membranes while NAD+ enhances energy production
Synergistic
MOTS-cBoth are mitochondrial peptides - MOTS-c regulates metabolism while SS-31 protects mitochondrial structure
Synergistic
HumaninAnother mitochondrial-derived peptide - may work together to protect cellular function and reduce apoptosis
Compatible
GHK-CuDifferent mechanisms - GHK-Cu works on gene expression while SS-31 directly protects mitochondria
Compatible
BPC-157No known interactions - BPC-157 focuses on tissue repair while SS-31 targets cellular energetics
Compatible
Thymosin Beta-4Both have cardioprotective effects through different pathways - may be complementary

Safety notes

Excellent safety profile in clinical trials

No significant side effects reported at therapeutic doses

May cause mild injection site reactions

Monitor for allergic reactions (very rare)

Safe for long-term use based on current data

No known drug interactions

Research studies

Barth Syndrome Clinical Trial - FDA Approval (2025)

Human | 40mg daily | Phase 3 | FDA Approved

Led to FDA accelerated approval of FORZINITY (elamipretide) in September 2025 - the first mitochondrial-targeting therapy approved. Indicated for improving muscle strength in Barth syndrome patients ≥30kg.

Phase 2 Trial for Primary Mitochondrial Myopathy (2018)

Human | 40mg daily | 12 weeks | 36 patients

Showed significant improvements in 6-minute walk test and fatigue scores in patients with mitochondrial myopathies, with excellent safety profile and no serious adverse events.

Cardioprotection in Heart Failure Model (2019)

Human | 0.25mg/kg/hr infusion | During cardiac surgery

Reduced cardiac injury markers and improved mitochondrial function in patients undergoing cardiac surgery, suggesting powerful cardioprotective effects.

Age-Related Muscle Dysfunction Study (2020)

Animal model | Various doses | 8 weeks | Aged mice

Reversed age-related decline in muscle function, increased ATP production, and reduced oxidative stress in skeletal muscle of aged mice.